To avoid misunderstanding patients with liver cirrhosis Nutrition
Suffering from “cirrhosis,” Aunt Lee was discharged to go home last week, the whole family to take care of her carefully, filial daughter, specially elaborate turtle stew of black-bone chicken soup added to her body. I did not expect at noon eating a meal in the evening on the minds of confused Aunt Lee, and distinguish between daughter and wife. To the next morning, Auntie Lee suddenly a coma.
In the hospital, the doctors informed of the critically ill and told the families: That was Aunt Lee cirrhosis of the liver to make up into hepatic coma! Is this how the matter?
Why the “hepatic coma”
The problem lies in the original Aunt Lee filial daughter to the mother’s black-bone chicken turtle soup.
Although the liver is the body’s metabolism and detoxification center, but with decompensated cirrhosis (cirrhosis of the liver in the late stage) patients, metabolism and detoxification of their liver dysfunction, for ammonia, mercaptans and other toxic substances in the body metabolism decreased significantly. Consumption of animal high-protein diet after the intestinal production of ammonia and mercaptan significantly increased, while the hardening of the liver at this time but can not be like normal liver of such toxic substances as the rapid metabolism, clearance, leading to such toxic substances in the body concentration increased, thus causing brain dysfunction, that is, medical-called “hepatic encephalopathy”, also known as “hepatic coma.” Aunt Lee is because a good appetite, eating a lot of her well-simmered stew of black-bone chicken turtle soup such a high-protein diet, it soon appeared hepatic coma.
“Hepatic coma,” the performance of
Hepatic encephalopathy is the most serious complications of cirrhosis of the liver, but it is the condition of performance is often overlooked.
Mild hepatic encephalopathy due to no obvious clinical manifestations have been treated as normal, taking part in normal social activities, and can demonstrate the abnormal character and behavior, such as the recently demonstrated a sudden excitement, aggressive, or indifferent, selfish , or in driving the risk, means of transport accidents. If timely medical treatment, and further development could demonstrate the time, place and close relatives of cognitive dysfunction, such as Auntie Lee after eating black-bone chicken turtle soup goes as far as her daughter recognized as a wife. If the disease further development, and may be manifested as drowsiness or no perception of the coma, when there is life in danger.
Patients with liver cirrhosis How to tonic
Cirrhosis patients nursed back to health and nutritional supplements in daily, it should be carefully noted and have a choice.
First, avoid eating high-protein diet, not to the human gut ammonia production surge.
Secondly, in particular, not to eat large amounts of animal protein. In addition to an increase in ammonia production outside the animal protein metabolites contain more aromatic amino acids, such amino acids can inhibit nerve conduction in the liver cirrhosis induced by hepatic coma.
Third, patients with decompensated cirrhosis of the liver a small amount of edible protein suitable. Because the plant protein in the aromatic amino acid less, branched-chain amino acids have been more inclined, while the branched-chain amino acids can be antagonistic effect of some toxic substances on brain function block.
Fourth, it can eat bananas and other fruits to keep stool smooth, 1-2 times a day, and always maintain the intestinal production of ammonia on a timely basis.
Fifth, in loss of appetite, or vomiting, diarrhea, you should promptly potassium, such as the consumption of fresh cucumber juice, apple juices to avoid hypokalemia alkalosis caused by hepatic encephalopathy.
Sixth, appropriate vitamin supplements and probiotics, such as vitamin C, vitamin B2, vitamin K, such as Lactobacillus acidophilus and stable environment for the body.
Seventh, unless the bleeding obvious after the anemia, or generally avoided in patients with cirrhosis of the nutritional or iron preparations containing minerals, because of increased hepatic iron has a hardening effect.
Eighth, as far as possible avoid the use of sedative sleep class of drugs to avoid this direct-induced hepatic coma.
Ninth, have esophageal varices, usually small rotten food should be doing more, avoid eating too coarse food, is strictly prohibited eating hard barbed type of food, such as barbed fish, chicken bones, as well as nuts and so on, to prevent scratch varicose veins of the esophagus or gastric vein, leading to upper gastrointestinal bleeding.