Fatty liver is the key to psychological prevention
Given the current anti-obesity effects no method of treatment of the existing measures are unsatisfactory, but the threat of obesity on human health, a huge and should therefore be particular emphasis on the prevention of overweight and obesity.
From the mother during pregnancy should be initiated to prevent, not to over-nutrition to the fetus and mother overweight. After birth, to breast-feeding, on-demand breast-feeding, do not demand that children eat too much, in particular, should be corrected hope that the more fat the better the idea of children from childhood to develop good eating habits, eat candy, cakes and other sweet snacks and dried foods nature, not eating and drinking, and to develop the habit of loving exercise to prevent the pre-puberty and puberty obesity. Middle age and menopause, should be adjusted according to the specific amount of food and food structure, and enhance the exercise, to keep the heat out of balance. After retirement should be an appropriate reduction in food intake, in particular, should be reduced by high-fat, high-calorie diet, eat fruits and vegetables, do not eat, or eat animal intestines, eat less salt, amount of tea, and cultivate the habit of hyperactivity, as far as possible to extend the Daily participation in sports time.
As the person’s life can occur at any time in obesity, therefore, the prevention of obesity should be long-term, enduring task. Scientific and rational food system, and promptly correct unhealthy eating and living habits, as well as long-standing moderate amount of physical activity to prevent obesity and its associated complications in an effective manner.
How to prevent toxic fatty liver
For industrial workers, the occupational exposure to the liver clear of toxic substances and potentially toxic substances occurring non-alcoholic steatohepatitis is one important reason.
Environment of the liver toxic substances include: mineral, the product of industrial processes, and the nature (plants, fungi, bacteria) exist in a variety of pro-liver poison. Benzene, ethylene dichloride, ethylene dichloride, barium salts, and cerium can cause isolated liver steatosis. Inorganic arsenic compounds, bromobenzene, carbon tetrachloride, chloroform, chlorine biphenyl, polychlorinated naphthalene, methylene chloride, 2 nitrobenzene, trinitrotoluene, tetrachloroethane, ethylene dichloride, naphthalene, 22 3, phosphorus, chromium, can cause liver cell necrosis and fatty degeneration.
The same as with other occupational poisoning, toxic fatty liver prevention include improving sanitation, improve operational processes and strengthen personal protection and so on. Specific measures: ① pre-employment health checks should include liver function, and HBsAg, should be considered abnormal liver poison can not engage in contact with the work of the pro-; ② pro-liver toxic exposure of workers to be medically examined on a regular basis liver function and liver B-, abnormal should be transferred and be properly handled; ③ contact with the pro-liver poison workers, should be disabled with liver damage, as well as on hepatic drug-metabolizing enzymes, such as mixed-function oxidases are induced by drugs, avoid contact with 223 other pesticides; ④ Liver toxic exposure of workers to be pro-temperance, dietary protein content, as well as the appropriate increase in vitamin B and vitamin C.
How to prevent drug-induced fatty liver
Several dozens of drugs may be related to fatty liver, such as the use of long-term high-dose corticosteroids, tetracycline, synthetic estrogen, 3-phenoxy alkyl, L-asparaginase, methotrexate, nifedipine, sodium valproate, as well as Amiodarone and other anti-arrhythmia drugs. Interestingly, chlorine Bedin fat (clofibrate), flexible enzyme cholesterol-lowering drugs not only is not an effective prevention and treatment of fatty liver, and sometimes contrary, it will trigger and exacerbate fatty liver. Therefore, in taking on the role of liver injury in drugs, medication is important to note the following points: ① must strictly abide by doctor’s advice, not hearsay, self-medication. Dosage should not be too big, not too many types of medication, in order to avoid drug interactions, respectively, Chinese and Western medicine is best to use, such as must be taken in the middle should interval 2 ~ 3 hours. Pairs of patients with multiple diseases, drug treatment, the focus should be phased medication. ② to observe the adverse drug reactions, physicians should be more exchange of information with patients, and if the long-term medication, should be regular follow-up liver function, blood lipids and liver B-for early detection of liver damage. Time out suspected drugs, most patients resume, only a very small occurrence of drug-induced liver disease become severe hepatitis or cirrhosis of the liver.
How to prevent total parenteral nutrition in patients with fatty liver
Total parenteral nutrition is supplied through the central venous catheter in patients with all the necessary nutrients so that patients do not eat in the state can maintain a good nutritional status. Used for high intestinal fistula, esophageal fistula, cancer patients before and after surgery and so on. Clinical study found that total parenteral nutrition can cause high blood sugar, high blood lipids, as well as the lack of essential fatty acids and other metabolic disorders. Adult given intravenous nutrition for more than two weeks, the liver biopsy can show liver steatosis and periportal cholestasis. Hepatic steatosis most vulnerable to the granting of a large number of glucose infusion rate over the liver oxidative capacity, resulting in increased synthesis of liver fat. Insulin and glucagon concentrations, especially in the portal vein insulin / glucagon ratio increases, is a key factor leading to fatty liver. Reasonable to give intravenous nutrition types, early resumption of oral intake of food or tube feeding diet, appropriate or necessary to increase with choline phospholipids (Essentiale) drugs such as parenteral nutrition to help prevent occurrence of fatty liver.
On how to prevent post-surgical patients with fatty liver
Intestinal diversion surgery, treatment of obesity, gastroplasty, biliopancreatic diversion surgery and extensive small bowel resection surgery may also cause fatty liver, increased liver fat deposition often occurred in the 6 months after surgery, after decreasing until the diversion surgery 2 ~ 3 years. It is primarily a passive use of fatty acids in adipose tissue, therefore, to these patients the blood essential amino acid content also decreased with protein-energy malnutrition, lack of similar findings, bacterial toxins and acid stones may also play negative role, perhaps interpretation associated with liver necrosis and fibrosis. Re-intestinal anastomosis to restore normal intestinal direction, and added essential amino acids, hepatic fat accumulation can subside.
In addition, post-surgical patients with high-fat, high-calorie diet, and limit activities, can cause short-term significant increase in body weight and lead to fatty liver. Therefore, the postoperative nutritional supplements should not over-appropriate exercises as soon as possible and postoperative care, to maintain smooth stool to facilitate the physical restoration and maintenance of ideal body weight, thus preventing excess nutrients such as fatty liver disease from occurring.